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Influenza epidemic of 1918 research paper
get enough samples of the virus to reproduce. Even harder hit were the Alaskan Inuit, with a death rate between 25 and 50 per cent: in some small Alaskan communities everybody died. These levels will produce hyperventilation in 33 of patients, as well as lung edema in 3 of patients. He used to talk with me about his favourite books and music, but for the last couple of years he had been confused and distracted. "Resistance to anti-influenza drugs: adamantanes and neuraminidase inhibitors". 49 As a result of this lack of antigenic diversity, a degree of immunity to influenza B is usually acquired at an early age. A b c d e Weber TP, Stilianakis NI (November 2008). 100 101 However, another possibility is that these large amounts of cytokines are just a result of the massive levels of viral replication produced by these strains, and the immune response does not itself contribute to the disease.
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36f "Bacterial Pneumonia Caused Most Deaths in 1918 Influenza Pandemic". According to the Odessan novelist Mendele Mocher Sforim, the bride and groom were the most frightful cripples, degraded paupers and lamentable neer-do-wells as were in the district. A large factor in the worldwide occurrence of this flu was increased travel. Cheung CY, Poon LL, Lau AS, Luk W, Lau YL, Shortridge KF, Gordon S, Guan Y, Peiris JS (December 2002). These sudden large changes allow the virus to infect new host species and quickly overcome protective immunity. Two kinds of protein jut out from its surface: Haemagglutinin is the skeleton key that allows the flu virus to slip into living cells; Neuraminidase is the battering-ram that bursts its progeny out. 85 Although a single sneeze releases up to 40,000 droplets, 87 most of these droplets are quite large and will quickly settle out of the air.
Curr Top Microbiol Immunol. 17 Symptoms of influenza can start quite suddenly one to two days after infection.
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